| May 17, 2004 | |||||||||
In Print![]() Photo of Haber by Steve Gilbert/Johnson courtesy of Bruce Johnson
From Focus: The question of why the targeted cancer drug gefitinib (Iressa) is spectacularly effective on 10 percent of lung cancers while leaving many untouched has been answered by two independent HMS studies. They found that mutations in gefitinib's target receptor determine whether tumors respond to the drug or not. Senior authors, including Daniel Haber (left) and Bruce Johnson, say the work supports the general view that targeting mutations crucial for tumor growth may be an effective approach against cancer.
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HeadlinesQuality of Life Continues to Change Long-term After Treatment for Prostate Cancer
UpcomingResearch Seminar:Aquaporin Water Channels: Blueprint for a Molecular Plumbing System
Peter Agre, Nobel Laureate in Chemistry
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